1
Er) were crossed to Roc1aG1/FM7 mothers and scored for the presence of rescued Roc1aG1 males. While the wild type Roc1a transgene was able to rescue the lethality of the Roc1aG1 mutation, none of 5 different ANBR transgenic insertions were able to do so (Fig. 4A). This was not a result of expression level differences, because all 5 of the ANBR proteins accumulated to a level comparable to the cont